The IκB kinase complex is a regulator of mRNA stability

EMBO J. 2018 Dec 14;37(24):e98658. doi: 10.15252/embj.201798658. Epub 2018 Nov 22.

Abstract

The IκB kinase (IKK) is considered to control gene expression primarily through activation of the transcription factor NF-κB. However, we show here that IKK additionally regulates gene expression on post-transcriptional level. IKK interacted with several mRNA-binding proteins, including a Processing (P) body scaffold protein, termed enhancer of decapping 4 (EDC4). IKK bound to and phosphorylated EDC4 in a stimulus-sensitive manner, leading to co-recruitment of P body components, mRNA decapping proteins 1a and 2 (DCP1a and DCP2) and to an increase in P body numbers. Using RNA sequencing, we identified scores of transcripts whose stability was regulated via the IKK-EDC4 axis. Strikingly, in the absence of stimulus, IKK-EDC4 promoted destabilization of pro-inflammatory cytokines and regulators of apoptosis. Our findings expand the reach of IKK beyond its canonical role as a regulator of transcription.

Keywords: IKK; EDC4; P bodies; RNA stability; post‐transcriptional regulation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Endoribonucleases / genetics
  • Endoribonucleases / metabolism
  • HEK293 Cells
  • Hep G2 Cells
  • Humans
  • I-kappa B Kinase / genetics
  • I-kappa B Kinase / metabolism*
  • Multiprotein Complexes / genetics
  • Multiprotein Complexes / metabolism*
  • Proteins / genetics
  • Proteins / metabolism*
  • RNA Stability*
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism*
  • Trans-Activators / genetics
  • Trans-Activators / metabolism

Substances

  • EDC4 protein, human
  • Multiprotein Complexes
  • Proteins
  • RNA, Messenger
  • Trans-Activators
  • I-kappa B Kinase
  • IKBKB protein, human
  • Endoribonucleases
  • DCP1A protein, human
  • DCP2 protein, human