Bronchial epithelial cells of young and old mice directly regulate the differentiation of Th2 and Th17

Biosci Rep. 2019 Feb 1;39(2):BSR20181948. doi: 10.1042/BSR20181948. Print 2019 Feb 28.

Abstract

To determine whether or not house dust mite (HDM) and HDM+lipopolysaccharide (LPS) exposure causes a difference in T-cell subsets from young and old mice. The bronchial epithelial cells (BECs) from young and old mice were divided into three groups (PBS (control), HDM, and HDM+LPS). CD4+ naive T cells from the spleen and lymph nodes were collected after 24 h of co-culture with BECs. The number of Th2 and Th17 cells was elevated in the HDM and HDM+LPS groups compared with the control group; these responses were exacerbated when exposed to HDM+LPS. The number of HDM- and HDM+LPS-specific Th2/Th17 cells in young mice was higher than old mice; however, the Th2:Th17 cell ratio was greater in young mice, whereas the Th17:Th2 cell ratio was greater in old mice. The expression of GATA-3 and RORc was increased in the HDM+LPS and HDM groups compared with the PBS group and exhibited most in HDM+LPS group. The expression of HDM+LPS-specific GATA-3 in young mice was higher, while the expression of HDM+LPS-specific RORc in old mice was higher. Murine BECs directly regulated CD4+ naive T-cell differentiation under allergen exposure.

Keywords: Th17 cells; Th2 cells; asthma; epithelial cells.

Publication types

  • Comparative Study

MeSH terms

  • Age Factors
  • Animals
  • Asthma / immunology
  • Asthma / pathology*
  • Bronchi / cytology*
  • CD4-Positive T-Lymphocytes
  • Cell Differentiation / drug effects
  • Cells, Cultured
  • Epithelial Cells / drug effects
  • Epithelial Cells / immunology
  • Epithelial Cells / metabolism
  • Epithelial Cells / pathology
  • GATA3 Transcription Factor / metabolism
  • Lipopolysaccharides / toxicity
  • Male
  • Mice, Inbred C57BL
  • Nuclear Receptor Subfamily 1, Group F, Member 3 / metabolism
  • Pyroglyphidae
  • Th17 Cells / cytology*
  • Th17 Cells / immunology
  • Th2 Cells / cytology*
  • Th2 Cells / immunology

Substances

  • GATA3 Transcription Factor
  • Gata3 protein, mouse
  • Lipopolysaccharides
  • Nuclear Receptor Subfamily 1, Group F, Member 3
  • Rorc protein, mouse