Autocrine or paracrine models of cytokine production and utilization in B-cell chronic lymphocytic leukaemia

Nouv Rev Fr Hematol (1978). 1988;30(5-6):339-41.

Abstract

The possibility that autocrine or paracrine mechanisms of cytokine production and utilization may take place in patients with B-cell chronic lymphocytic leukaemia (B-CLL) is discussed. In particular, evidence is provided for a likely role of interleukin 2 (IL2) on the neoplastic B-cell compartment. Furthermore, the possibility that tumor necrosis factor (TNF) may also be involved is suggested.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Humans
  • Interleukin-2 / biosynthesis*
  • Interleukin-2 / physiology
  • Leukemia, Lymphocytic, Chronic, B-Cell / metabolism*
  • Models, Biological
  • Tumor Necrosis Factor-alpha / biosynthesis*
  • Tumor Necrosis Factor-alpha / physiology

Substances

  • Interleukin-2
  • Tumor Necrosis Factor-alpha