T follicular regulatory (Tfr) cells: Dissecting the complexity of Tfr-cell compartments

Immunol Rev. 2019 Mar;288(1):112-127. doi: 10.1111/imr.12739.

Abstract

Germinal centers (GC) have been known as key anatomic structures in humoral immunity, where isotype switching and affinity maturation occur. As a consequence, elucidation of GC regulation has potential implications for the understanding of autoantibody-mediated diseases. It is now accepted that different regulatory mechanisms coexist, including the action of a specialized population of Foxp3+ regulatory T cells with unique access to the B-cell follicle: the T follicular regulatory (Tfr) cells. Tfr cells develop through a multistep process requiring migration through different compartments of lymphoid tissues. This review discusses the ontogeny and physiology of Tfr cells, their distribution within distinct anatomic compartments, and their function. A greater understanding of Tfr biology and GC regulation is likely to lead to better stratification of patients with autoantibody-mediated diseases, and to the identification of novel therapeutic targets.

Keywords: Foxp3; T follicular helper cells; T follicular regulatory cells; T regulatory cells; humoral responses; interleukin-2.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Adaptive Immunity
  • Animals
  • B-Lymphocytes / immunology*
  • Cell Differentiation
  • Cell Movement
  • Forkhead Transcription Factors / metabolism
  • Germinal Center / immunology*
  • Humans
  • Lymphocyte Activation
  • T-Lymphocytes, Helper-Inducer / immunology*
  • T-Lymphocytes, Regulatory / immunology*

Substances

  • FOXP3 protein, human
  • Forkhead Transcription Factors