Oral L-glutamine pretreatment attenuates skeletal muscle atrophy induced by 24-h fasting in mice

J Nutr Biochem. 2019 Aug:70:202-214. doi: 10.1016/j.jnutbio.2019.05.010. Epub 2019 May 25.

Abstract

L-Glutamine (L-Gln) supplementation has been pointed out as an anticatabolic intervention, but its effects on protein synthesis and degradation signaling in skeketal muscle are still poorly known. The effects of L-Gln pretreatment (1 g kg-1 day-1 body weight for 10 days) on muscle fiber cross-sectional area (CSA), amino acid composition (measured by LC-MS/MS) and protein synthesis (Akt-mTOR) and degradation (ubiquitin ligases) signaling in soleus and extensor digitorum longus (EDL) muscles in 24-h-fasted mice were investigated. The fiber CSA of EDL muscle was not different between the L-Gln-fasted and L-Gln-fed groups. This finding was associated with reduced contents of L-Leu and L-Iso and activation of protein synthesis signaling (p-RPS6Ser240/244 and Akt-mTOR). The spectrum of soleus muscle fiber CSA distribution was larger in L-Gln-fasted as compared with placebo-fasted mice. This effect of L-Gln pretreatment was associated with changes in red fibers L-Gln metabolism as indicated by increased intracellular L-glutamine/L-glutamate ratio, L-aspartate and GABA levels. L-Gln supplementation reduced fasting-induced mass loss in tibialis anterior and gastrocnemius muscles. Evidence is presented that pretreatment with L-glutamine attenuates skeletal muscle atrophy induced by 24-h fasting through mechanisms that vary with the muscle fiber type.

Keywords: 4E-BP1; GABA; L-Glutamate; Muscle fiber type; RPS6.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing / metabolism
  • Adipose Tissue / metabolism
  • Administration, Oral
  • Animals
  • Cell Cycle Proteins / metabolism
  • Fasting / adverse effects*
  • Glutamine / administration & dosage*
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Muscle, Skeletal / pathology*
  • Muscular Atrophy / prevention & control*
  • Ribosomal Protein S6 / metabolism
  • Signal Transduction
  • gamma-Aminobutyric Acid / metabolism

Substances

  • Adaptor Proteins, Signal Transducing
  • Cell Cycle Proteins
  • Eif4ebp1 protein, mouse
  • Ribosomal Protein S6
  • ribosomal protein S6, mouse
  • Glutamine
  • gamma-Aminobutyric Acid