Metformin reduces c-Fos and ATF3 expression in the dorsal root ganglia and protects against oxaliplatin-induced peripheral sensory neuropathy in mice

Neurosci Lett. 2019 Sep 14:709:134378. doi: 10.1016/j.neulet.2019.134378. Epub 2019 Jul 17.

Abstract

Oxaliplatin is a third-generation platinum drug commonly used as the first line treatment of metastatic colorectal cancer. Oxaliplatin-based anticancer regimens course with dose-limiting neurotoxicity. The pharmacological strategies used to manage such side effect are not totally effective. Metformin is an anti-diabetic drug that is described to negatively modulate painful diabetic neuropathy. Then, this study aimed to assess the effect of metformin in the oxaliplatin-induced peripheral sensory neuropathy in mice. For that purpose, Swiss male mice were injected with oxaliplatin (1, 2 or 4 mg/kg, i.v., twice a week with a total of nine injections) alone or in combination with daily administration of metformin (250 mg/kg, p.o.). Thermal and mechanical nociceptive tests were performed once a week for five weeks. Then, the animals were euthanized on day 35 post-first injection of oxaliplatin and the dorsal root ganglia were harvested for the assessment of c-Fos and ATF3 expressions. Oxaliplatin caused a nociceptive response accompanied by the increased expression of c-Fos and ATF3 in the dorsal root ganglia and spinal cord. In addition, the oxaliplatin-associated nociception was significantly attenuated by metformin (P < 0.05), which also reduced the expression of c-Fos and ATF3 (P < 0.05). Therefore, metformin protected from the peripheral sensory neuropathy induced by oxaliplatin, which was confirmed by the reduction of c-Fos and ATF3 expression, two known neuronal activation and damage markers, respectively.

Keywords: Metformin; Neurotoxicity; Oxaliplatin.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Activating Transcription Factor 3 / antagonists & inhibitors*
  • Activating Transcription Factor 3 / biosynthesis
  • Activating Transcription Factor 3 / genetics
  • Animals
  • Antineoplastic Agents / toxicity
  • Ganglia, Spinal / drug effects
  • Ganglia, Spinal / metabolism*
  • Gene Expression
  • Hypoglycemic Agents / pharmacology
  • Hypoglycemic Agents / therapeutic use
  • Male
  • Metformin / pharmacology
  • Metformin / therapeutic use*
  • Mice
  • Oxaliplatin / toxicity*
  • Peripheral Nervous System Diseases / drug therapy
  • Peripheral Nervous System Diseases / genetics
  • Peripheral Nervous System Diseases / metabolism*
  • Proto-Oncogene Proteins c-fos / antagonists & inhibitors*
  • Proto-Oncogene Proteins c-fos / biosynthesis
  • Proto-Oncogene Proteins c-fos / genetics

Substances

  • Activating Transcription Factor 3
  • Antineoplastic Agents
  • Atf3 protein, mouse
  • Hypoglycemic Agents
  • Proto-Oncogene Proteins c-fos
  • Oxaliplatin
  • Metformin