[Hereditary angioedema]

Internist (Berl). 2019 Sep;60(9):987-995. doi: 10.1007/s00108-019-0644-1.
[Article in German]

Abstract

Hereditary angioedema (HAE) encompasses a heterogeneous group of diseases with similar phenotypes but different underlying genotypes. Specific clinical signs may point to HAE as opposed to histaminergic angioedema: the typical prolonged development of angioedema over time, positive family history, a lack of response to antihistamines and steroids and response to bradykinin antagonists are typical signs of HAE. The different types of HAE may be associated with a severe clinical course. They are life-long conditions and are still potentially life-threatening. The quality of life of patients with HAE may be considerably impaired. Management plans should be individualized, which is facilitated by the variety of specific medicastions available.

Keywords: Bradykinin; Complement C1 inhibitor protein; Factor XII; Hereditary angioedema; Prophylaxis, angioedema.

Publication types

  • Review

MeSH terms

  • Angioedema / diagnosis
  • Angioedema / physiopathology
  • Angioedemas, Hereditary / diagnosis
  • Angioedemas, Hereditary / genetics*
  • Angioedemas, Hereditary / physiopathology
  • Bradykinin* / metabolism
  • Complement C1 Inhibitor Protein*
  • Factor XII* / genetics
  • Factor XIIa* / metabolism
  • Humans
  • Mutation
  • Quality of Life

Substances

  • Complement C1 Inhibitor Protein
  • Factor XII
  • Factor XIIa
  • Bradykinin