Rapid onset type-1 diabetes and diabetic ketoacidosis secondary to nivolumab immunotherapy: a review of existing literature

BMJ Case Rep. 2019 Aug 26;12(8):e229568. doi: 10.1136/bcr-2019-229568.

Abstract

Nivolumab is a programmed cell death receptor (PD-1) inhibitor that is increasingly used for various malignancies, both as a first line agent and as salvage therapy. Being a PD-1/PD-1 ligand checkpoint inhibitor, it is known to cause autoimmune inflammation of various organs and has been associated with thyroiditis, insulitis, colitis, hepatitis and encephalitis to name a few. There are increasing reports of nivolumab leading to acute onset fulminant type 1 diabetes and diabetic ketoacidosis (DKA). We present a case of a 68-year-old man who developed DKA after 2 doses of nivolumab for metastatic melanoma. He was found to have type 1 diabetes, but no diabetes related antibodies were positive. He recovered from diabetes and continues to use insulin 1 year after his diagnosis. This case and associated review illustrates the importance of educating and monitoring patients who start nivolumab therapy regarding this potentially life threatening complication.

Keywords: chemotherapy; diabetes; immunological products and vaccines.

Publication types

  • Case Reports
  • Review

MeSH terms

  • Aged
  • Antineoplastic Agents, Immunological / administration & dosage
  • Antineoplastic Agents, Immunological / adverse effects
  • Antineoplastic Agents, Immunological / immunology
  • Diabetes Mellitus, Type 1* / complications
  • Diabetes Mellitus, Type 1* / diagnosis
  • Diabetes Mellitus, Type 1* / drug therapy
  • Diabetes Mellitus, Type 1* / etiology
  • Diabetic Ketoacidosis* / etiology
  • Diabetic Ketoacidosis* / therapy
  • Drug Monitoring / methods
  • Humans
  • Hypoglycemic Agents / administration & dosage
  • Insulin / administration & dosage*
  • Male
  • Melanoma* / drug therapy
  • Melanoma* / pathology
  • Neoplasm Staging
  • Nivolumab* / administration & dosage
  • Nivolumab* / adverse effects
  • Nivolumab* / immunology
  • Programmed Cell Death 1 Receptor / antagonists & inhibitors*
  • Treatment Outcome

Substances

  • Antineoplastic Agents, Immunological
  • Hypoglycemic Agents
  • Insulin
  • Programmed Cell Death 1 Receptor
  • Nivolumab