Association between FSAP 1601G > A polymorphism and venous thromboembolism risk: A meta-analysis

Phlebology. 2020 Jun;35(5):345-353. doi: 10.1177/0268355519881604. Epub 2019 Oct 21.

Abstract

Objective: Previous several studies have shown that factor VII-activating protease (FSAP) gene 1601G>A polymorphism is related to the occurrence of venous thromboembolism, but the results are inconsistent and controversial. Therefore, we conducted a meta-analysis to explore the association between FSAP 1601G>A polymorphism and venous thromboembolism susceptibility.

Methods: We managed a systematic literature search through Pubmed, Embase, Web of Science, China National Knowledge Infrastructure, and WanFang databases to collect research data related to FSAP gene 1601G>A polymorphism and susceptibility to venous thromboembolism published before May 2019. Data analysis was performed through Revman 5.3 and Stata 12.0 software, the pooled odd ratios and 95% confidence intervals were calculated. Additionally, the sensitivity analysis and publication bias assessment were also performed.

Results: A total of seven case-control studies were included and evaluated, including 2411 venous thromboembolism cases and 2850 controls. The meta-analysis results revealed that the FSAP 1601G>A mutation is associated with venous thromboembolism risk, and statistically significance was observed under three genetic comparison models (A: G, odds ratio: 1.33, 95% confidence interval: 1.07-1.66; GA: GG, odds ratio: 1.34, 95% confidence interval: 1.06-1.68; and GA + AA: GG, odds ratio: 1.33, 95% confidence interval: 1.06-1.66).

Conclusion: This study demonstrated that the FSAP 1601G>A polymorphism may be associated with venous thromboembolism susceptibility.

Keywords: Factor VII-activating protease; meta-analysis; polymorphism; venous thromboembolism.

Publication types

  • Meta-Analysis
  • Systematic Review

MeSH terms

  • Case-Control Studies
  • Genetic Association Studies
  • Genetic Predisposition to Disease
  • Humans
  • Phenotype
  • Polymorphism, Genetic*
  • Risk Assessment
  • Risk Factors
  • Serine Endopeptidases / genetics*
  • Venous Thromboembolism / diagnosis
  • Venous Thromboembolism / genetics*

Substances

  • HABP2 protein, human
  • Serine Endopeptidases