Reconstituting the Mammalian Apoptotic Switch in Yeast

Genes (Basel). 2020 Jan 29;11(2):145. doi: 10.3390/genes11020145.

Abstract

Proteins of the Bcl-2 family regulate the permeabilization of the mitochondrial outer membrane that represents a crucial irreversible step in the process of induction of apoptosis in mammalian cells. The family consists of both proapoptotic proteins that facilitate the membrane permeabilization and antiapoptotic proteins that prevent it in the absence of an apoptotic signal. The molecular mechanisms, by which these proteins interact with each other and with the mitochondrial membranes, however, remain under dispute. Although yeast do not have apparent homologues of these apoptotic regulators, yeast cells expressing mammalian members of the Bcl-2 family have proved to be a valuable model system, in which action of these proteins can be effectively studied. This review focuses on modeling the activity of proapoptotic as well as antiapoptotic proteins of the Bcl-2 family in yeast.

Keywords: BH3-only proteins; Bax; Bcl-2 protein family; Bcl-XL; Saccharomyces cerevisiae; apoptosis; mitochondria.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Apoptosis
  • Mitochondrial Membranes / metabolism*
  • Proto-Oncogene Proteins c-bcl-2 / metabolism*
  • Saccharomyces cerevisiae / metabolism
  • Saccharomyces cerevisiae / physiology*
  • Saccharomyces cerevisiae Proteins / metabolism

Substances

  • Proto-Oncogene Proteins c-bcl-2
  • Saccharomyces cerevisiae Proteins