Regulation of microRNAs by IRE1α in apoptosis: implications for the pathomechanism of neurodegenerative diseases

Int J Neurosci. 2020 Dec;130(12):1230-1236. doi: 10.1080/00207454.2020.1730833. Epub 2020 Feb 18.

Abstract

Although there are large differences in clinical and pathological features, age-related neurodegenerative diseases (NDs) share common pathogenetic mechanisms involving aggregation and deposition of misfolded proteins, which leads to progressive dysfunction and death of neurons. Up to now, it seems that apoptosis is one major form of neuronal cell death. This review provides an overview of recent progress in unfolded protein response (UPR) during apoptosis induced by abnormal protein aggregation and emphasizes on the potential role of inositol requiring enzyme 1 alpha (IRE1α)-microRNAs (miRNAs) mediated apoptosis in NDs, which will provide new insights in the pathogenesis of neurodegenerative diseases and novel therapeutic targets for the treatment of NDs.

Keywords: Apoptosis; IRE1α; microRNA (miRNA); misfolded proteins; neurodegenerative diseases (NDs).

Publication types

  • Review

MeSH terms

  • Apoptosis / physiology*
  • Endoribonucleases / metabolism*
  • Humans
  • MicroRNAs / metabolism*
  • Neurodegenerative Diseases / enzymology
  • Neurodegenerative Diseases / metabolism*
  • Neurodegenerative Diseases / pathology*
  • Protein Serine-Threonine Kinases / metabolism*
  • Unfolded Protein Response / physiology*

Substances

  • MicroRNAs
  • ERN1 protein, human
  • Protein Serine-Threonine Kinases
  • Endoribonucleases