The role of mTOR signalling pathway in hypoxia-induced cognitive impairment

Int J Neurosci. 2021 May;131(5):482-488. doi: 10.1080/00207454.2020.1746308. Epub 2020 Apr 2.

Abstract

Hypoxia has been associated with cognitive impairment. Many studies have investigated the role of mTOR signalling pathway in cognitive functions but its role in hypoxia-induced cognitive impairment remains controversial. This review aimed to elucidate the role of mTOR in the mechanisms of cognitive impairment that may pave the way towards the mechanistic understanding and therapeutic intervention of hypoxia-induced cognitive impairment. mTORC1 is normally regulated during mild or acute hypoxic exposure giving rise to neuroprotection, whereas it is overactivated during severe or chronic hypoxia giving rise to neuronal cells death. Thus, it is worth exploring the possibility of maintaining normal mTORC1 activity and thereby preventing cognitive impairment during severe or chronic hypoxia.

Keywords: BDNF; HIF-1; Hypoxia; cognitive impairment; mTORC1; mTORC2.

Publication types

  • Review

MeSH terms

  • Animals
  • Brain-Derived Neurotrophic Factor / metabolism*
  • Cognitive Dysfunction / etiology
  • Cognitive Dysfunction / metabolism*
  • Cognitive Dysfunction / prevention & control
  • Humans
  • Hypoxia / complications
  • Hypoxia / metabolism*
  • Hypoxia-Inducible Factor 1, alpha Subunit / metabolism*
  • Mechanistic Target of Rapamycin Complex 1 / metabolism*
  • Mechanistic Target of Rapamycin Complex 2 / metabolism*
  • Signal Transduction / physiology*
  • TOR Serine-Threonine Kinases / metabolism*

Substances

  • Brain-Derived Neurotrophic Factor
  • Hypoxia-Inducible Factor 1, alpha Subunit
  • Mechanistic Target of Rapamycin Complex 1
  • Mechanistic Target of Rapamycin Complex 2
  • TOR Serine-Threonine Kinases