Skin Mycobiome of Psoriasis Patients is Retained during Treatment with TNF and IL-17 Inhibitors

Int J Mol Sci. 2020 May 29;21(11):3892. doi: 10.3390/ijms21113892.

Abstract

Background: Biological treatment relieves refractory skin lesions in patients with psoriasis; however, changes in the fungal microbiome (the mycobiome) on the skin are unclear.

Methods: The skin mycobiome of psoriasis patients treated with TNF inhibitors (TNFi, n = 5) and IL-17 inhibitors (IL-17i, n = 7) was compared with that of patients not receiving systemic therapy (n = 7). Skin swab samples were collected from non-lesional post-auricular areas. Fungal DNA was sequenced by ITS1 metagenomic analysis and taxonomic classification was performed.

Results: An average of 37543 reads/sample were analyzed and fungi belonging to 31 genera were detected. The genus Malassezia accounted for >90% of reads in 7/7 samples from the no-therapy group, 4/5 from the TNFi group, and 5/7 from the IL-17i group. Biodiversity was low in those three groups. Few members of the genus trichophyton were detected; the genus Candida was not detected at all. Among the Malassezia species, M. restricta was the major species in 6/7 samples from the no-therapy group, 4/5 from the TNFi group, and 5/7 from the IL-17i group whose the other largest species revealed M. globosa.

Conclusions: The mycobiome is retained on post-auricular skin during systemic treatment with TNF and IL-17 inhibitors.

Keywords: IL-17 inhibitor; TNF inhibitor; biologics; fungal microbiome; mycobiome; psoriasis.

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Biodiversity
  • Biological Products
  • DNA, Fungal / genetics
  • DNA, Intergenic / genetics
  • Female
  • Humans
  • Interleukin-17 / antagonists & inhibitors*
  • Malassezia
  • Male
  • Metagenomics
  • Middle Aged
  • Mycobiome*
  • Pore Forming Cytotoxic Proteins / pharmacology
  • Psoriasis / drug therapy*
  • Psoriasis / microbiology*
  • Skin / microbiology*
  • Tumor Necrosis Factor Inhibitors / therapeutic use*
  • Young Adult

Substances

  • Biological Products
  • DNA, Fungal
  • DNA, Intergenic
  • Interleukin-17
  • Pore Forming Cytotoxic Proteins
  • Tumor Necrosis Factor Inhibitors