Response of resident murine peritoneal macrophages to in vivo administration of granulocyte-macrophage colony-stimulating factor

J Immunol. 1988 Mar 15;140(6):1910-5.

Abstract

The effect of s.c. inoculation of purified recombinant derived granulocyte-macrophage (GM)-CSF on resident murine peritoneal macrophages was assessed in this study. From 18 to 24 h after s.c. administration of GM-CSF to normal mice, the resident peritoneal macrophages were harvested and the levels of membrane-bound IL-1, FcR, Mac-1 cell-surface Ag, and class II MHC expression were assessed. Peritoneal cells from GM-CSF-inoculated mice had significantly greater levels of membrane-bound IL-1 than did control mice. In addition when resident peritoneal macrophages from normal mice were purified by adherence and grown in the presence of GM-CSF, they produced greater levels of both membrane-bound and secreted IL-1. The peritoneal cells from GM-CSF-inoculated mice did not differ from controls in the expression of class II MHC-encoded Ag. This observation was confirmed by the finding that GM-CSF was unable to induce class II MHC expression on P388D1 cells, whereas a secondary mixed leukocyte culture supernatant was. Peritoneal cells from GM-CSF-inoculated mice also exhibited greater levels of expression of FcR and the Mac-1 cell-surface Ag. This resulted in an increase in their ability to phagocytose opsonized SRBC in vitro.

MeSH terms

  • Animals
  • Antigens, Surface / biosynthesis
  • Colony-Stimulating Factors / pharmacology*
  • Female
  • Gene Expression Regulation / drug effects
  • Histocompatibility Antigens Class II / analysis
  • Interleukin-1 / biosynthesis
  • Macrophage-1 Antigen
  • Macrophages / analysis
  • Macrophages / drug effects*
  • Mice
  • Mice, Inbred C57BL
  • Peritoneal Cavity
  • Receptors, Fc / biosynthesis
  • Recombinant Proteins / pharmacology
  • Stimulation, Chemical

Substances

  • Antigens, Surface
  • Colony-Stimulating Factors
  • Histocompatibility Antigens Class II
  • Interleukin-1
  • Macrophage-1 Antigen
  • Receptors, Fc
  • Recombinant Proteins