Delayed rejection of heart allografts after extracorporeal donor-specific liver hemoperfusion. Role of Kupffer cells

Transplantation. 1988 Mar;45(3):628-32. doi: 10.1097/00007890-198803000-00025.

Abstract

Liver allografts have a privileged status in regard to acute rejection. In this experimental study, we have analyzed the immunosuppressive effects of an extracorporeal liver hemoperfusion. In the LEW-to-BN combination of inbred rats, donor-specific liver hemoperfusion can significantly delay acute rejection of heart allografts. Analysis of the immunological status of these animals revealed a significant decrease in donor-specific lymphocytotoxic antibodies and in cytotoxic T lympholysis. Reactivity in mixed lymphocyte culture was normal. After third-party (DA) liver hemoperfusion or after donor-specific (LEW) splenic hemoperfusion, prolongation of heart allograft survival was moderate. Previous blockade of Kupffer cells suppressed the effects of donor-specific liver hemoperfusion. These results suggest that the sequestration by Kupffer cells of a clone of cytotoxic T cells and/or lymphocytotoxic antibodies may explain the immunosuppressive effects of donor-specific liver hemoperfusion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibody Formation
  • Antibody Specificity
  • Cytotoxicity, Immunologic
  • Graft Rejection
  • Graft Survival
  • Heart Transplantation*
  • Hemoperfusion
  • Immunity, Cellular
  • Kupffer Cells / physiology*
  • Liver / immunology*
  • Rats
  • Rats, Inbred BN
  • Rats, Inbred Lew
  • Rats, Inbred Strains
  • Tissue Donors
  • Transplantation, Homologous