Abstract
In this article, we report our efforts towards improving in vitro human clearance in a series of 5-azaquinazolines through a series of C4 truncations and C2 expansions. Extensive DMPK studies enabled us to tackle high Aldehyde Oxidase (AO) metabolism and unexpected discrepancies in human hepatocyte and liver microsomal intrinsic clearance. Our efforts culminated with the discovery of 5-azaquinazoline 35, which also displayed exquisite selectivity for IRAK4, and showed synergistic in vitro activity against MyD88/CD79 double mutant ABC-DLBCL in combination with the covalent BTK inhibitor acalabrutinib.
Keywords:
5-Azaquinazoline; Aldehyde oxidase; DLBCL; IRAK4.
Copyright © 2020. Published by Elsevier Ltd.
MeSH terms
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Aldehyde Oxidase / metabolism
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Animals
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Binding Sites
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Cell Line, Tumor
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Cell Survival / drug effects
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Crystallography, X-Ray
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Dogs
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Drug Stability
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Half-Life
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Hepatocytes / metabolism
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Humans
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Interleukin-1 Receptor-Associated Kinases / antagonists & inhibitors*
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Interleukin-1 Receptor-Associated Kinases / metabolism
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Mice
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Microsomes, Liver / metabolism
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Molecular Dynamics Simulation
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Protein Kinase Inhibitors / chemistry
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Protein Kinase Inhibitors / metabolism*
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Protein Kinase Inhibitors / pharmacology
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Quinazolines / chemistry*
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Quinazolines / metabolism
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Quinazolines / pharmacology
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Rats
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Structure-Activity Relationship
Substances
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Protein Kinase Inhibitors
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Quinazolines
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Aldehyde Oxidase
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IRAK4 protein, human
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Interleukin-1 Receptor-Associated Kinases