A New Peracetylated Oleuropein Derivative Ameliorates Joint Inflammation and Destruction in a Murine Collagen-Induced Arthritis Model via Activation of the Nrf-2/Ho-1 Antioxidant Pathway and Suppression of MAPKs and NF-κB Activation

Nutrients. 2021 Jan 22;13(2):311. doi: 10.3390/nu13020311.

Abstract

: Oleuropein (OL), an olive tree secoiridoid and its peracetylated derivate (Per-OL) have exhibited several beneficial effects on LPS-stimulated macrophages and murine experimental systemic lupus erythematosus (SLE). This study was designed to evaluate dietary Per-OL in comparison with OL supplementation effects on collagen-induced arthritis (CIA) murine model. Three-weeks-old DBA-1/J male mice were fed from weaning with a standard commercial diet or experimental enriched-diets in 0.05 % (w/w) OL, 0.05% and 0.025% Per-OL. After six weeks of pre-treatment, arthritis was induced by bovine collagen type II by tail base injection (day 0) and on day 21, mice received a booster injection. Mice were sacrificed 42 days after the first immunization. Both Per-OL and OL diets significantly prevented histological damage and arthritic score development, although no statistically significant differences were observed between both compounds. Also, serum collagen oligomeric matrix protein (COMP), metalloprotease (MMP)-3 and pro-inflammatory cytokines levels were ameliorated in paws from secoiridoids fed animals. Mitogen-activated protein kinases (MAPK)s and nuclear transcription factor-kappa-B (NF-κB) activations were drastically down-regulated whereas nuclear factor E2-related factor 2 (Nrf2) and heme-oxygenase-1 (HO-1) protein expressions were up-regulated in those mice fed with OL and Per-OL diets. We conclude that both Per-OL and its parent compound, OL, supplements might provide a basis for developing a new dietary strategy for the prevention of rheumatoid arthritis.

Keywords: CIA; MAPK; NF-κB; Nrf2/HO1; arthritis; inflammation; oleuropein.

MeSH terms

  • Animals
  • Arthritis, Experimental / chemically induced
  • Arthritis, Experimental / diet therapy*
  • Arthritis, Experimental / metabolism
  • Dietary Supplements
  • Disease Models, Animal
  • Heme Oxygenase-1 / metabolism
  • Inflammasomes / drug effects*
  • Inflammasomes / metabolism
  • Iridoid Glucosides / pharmacology*
  • MAP Kinase Signaling System / drug effects
  • Male
  • Matrix Metalloproteinase 3 / metabolism
  • Membrane Proteins / metabolism
  • Mice
  • Mice, Inbred DBA
  • NF-E2-Related Factor 2 / metabolism
  • NF-kappa B / drug effects
  • Signal Transduction / drug effects

Substances

  • Inflammasomes
  • Iridoid Glucosides
  • Membrane Proteins
  • NF-E2-Related Factor 2
  • NF-kappa B
  • Nfe2l2 protein, mouse
  • oleuropein
  • Heme Oxygenase-1
  • Hmox1 protein, mouse
  • Matrix Metalloproteinase 3
  • Mmp3 protein, mouse