Pulmonary vascular proliferation in patients with severe COVID-19: an autopsy study

Thorax. 2021 Oct;76(10):1044-1046. doi: 10.1136/thoraxjnl-2020-216714. Epub 2021 Mar 23.

Abstract

Diffuse alveolar damage and thrombi are the most common lung histopathological lesions reported in patients with severe COVID-19. Although some studies have suggested increased pulmonary angiogenesis, the presence of vascular proliferation in COVID-19 lungs has not been well characterised. Glomeruloid-like microscopic foci and/or coalescent vascular proliferations measuring up to 2 cm were present in the lung of 14 out of 16 autopsied patients. These lesions expressed CD31, CD34 and vascular endothelial cadherin. Platelet-derived growth factor receptor-β immunohistochemistry and dual immunostaining for CD34/smooth muscle actin demonstrated the presence of pericytes. These vascular alterations may contribute to the severe and refractory hypoxaemia that is common in patients with severe COVID-19.

Keywords: ARDS; COVID-19; histology/cytology.

Publication types

  • Research Support, Non-U.S. Gov't
  • Comment

MeSH terms

  • Autopsy
  • COVID-19*
  • Cell Proliferation
  • Humans
  • Lung
  • SARS-CoV-2