Generation of caudal-type serotonin neurons and hindbrain-fate organoids from hPSCs

Stem Cell Reports. 2021 Aug 10;16(8):1938-1952. doi: 10.1016/j.stemcr.2021.06.006. Epub 2021 Jul 8.

Abstract

Serotonin (5-HT) neurons, the major components of the raphe nuclei, arise from ventral hindbrain progenitors. Based on anatomical location and axonal projection, 5-HT neurons are coarsely divided into rostral and caudal groups. Here, we propose a novel strategy to generate hindbrain 5-HT neurons from human pluripotent stem cells (hPSCs), which involves the formation of ventral-type neural progenitor cells and stimulation of the hindbrain 5-HT neural development. A caudalizing agent, retinoid acid, was used to direct the cells into the hindbrain cell fate. Approximately 30%-40% of hPSCs successfully developed into 5-HT-expressing neurons using our protocol, with the majority acquiring a caudal rhombomere identity (r5-8). We further modified our monolayer differentiation system to generate 5-HT neuron-enriched hindbrain-like organoids. We also suggest downstream applications of our 5-HT monolayer and organoid cultures to study neuronal response to gut microbiota. Our methodology could become a powerful tool for future studies related to 5-HT neurotransmission.

Keywords: hindbrain; human pluripotent stem cells; neuronal development; organoids; serotonin neurons.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Culture Techniques / methods*
  • Cell Differentiation / drug effects
  • Cell Differentiation / genetics
  • Cell Line
  • Humans
  • Immunohistochemistry / methods
  • Neural Stem Cells / cytology
  • Neural Stem Cells / metabolism
  • Neurogenesis / drug effects
  • Neurogenesis / genetics
  • Neurons / cytology*
  • Neurons / metabolism
  • Organoids / cytology*
  • Organoids / metabolism
  • Pluripotent Stem Cells / cytology*
  • Pluripotent Stem Cells / metabolism
  • Reproducibility of Results
  • Reverse Transcriptase Polymerase Chain Reaction / methods
  • Rhombencephalon / cytology*
  • Rhombencephalon / metabolism
  • Serotonin / metabolism*
  • Transcriptome / drug effects
  • Transcriptome / genetics
  • Tretinoin / pharmacology

Substances

  • Serotonin
  • Tretinoin