Early Inflammatory Cytokine Expression in Cerebrospinal Fluid of Patients with Spontaneous Intraventricular Hemorrhage

Biomolecules. 2021 Jul 30;11(8):1123. doi: 10.3390/biom11081123.

Abstract

We investigated cerebrospinal fluid (CSF) expression of inflammatory cytokines and their relationship with spontaneous intracerebral and intraventricular hemorrhage (ICH, IVH) and perihematomal edema (PHE) volumes in patients with acute IVH. Twenty-eight adults with IVH requiring external ventricular drainage for obstructive hydrocephalus had cerebrospinal fluid (CSF) collected for up to 10 days and had levels of interleukin-1α (IL-1α), IL-1β, IL-6, IL-8, IL-10, tumor necrosis factor-α (TNFα), and C-C motif chemokine ligand CCL2 measured using enzyme-linked immunosorbent assay. Median [IQR] ICH and IVH volumes at baseline (T0) were 19.8 [5.8-48.8] and 14.3 [5.3-38] mL respectively. Mean levels of IL-1β, IL-6, IL-10, TNF-α, and CCL2 peaked early compared to day 9-10 (p < 0.05) and decreased across subsequent time periods. Levels of IL-1β, IL-6, IL-8, IL-10, and CCL2 had positive correlations with IVH volume at days 3-8 whereas positive correlations with ICH volume occurred earlier at day 1-2. Significant correlations were found with PHE volume for IL-6, IL-10 and CCL2 at day 1-2 and with relative PHE at days 7-8 or 9-10 for IL-1β, IL-6, IL-8, and IL-10. Time trends of CSF cytokines support experimental data suggesting association of cerebral inflammatory responses with ICH/IVH severity. Pro-inflammatory markers are potential targets for injury reduction.

Keywords: cerebrospinal fluid; cytokines; intracerebral hemorrhage; intraventricular hemorrhage; neuroinflammation.

Publication types

  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Cerebral Intraventricular Hemorrhage / cerebrospinal fluid
  • Cerebral Intraventricular Hemorrhage / genetics*
  • Cerebral Intraventricular Hemorrhage / physiopathology
  • Cerebral Intraventricular Hemorrhage / therapy
  • Chemokine CCL2 / cerebrospinal fluid
  • Chemokine CCL2 / genetics
  • Drainage / methods
  • Female
  • Gene Expression*
  • Humans
  • Hydrocephalus / cerebrospinal fluid
  • Hydrocephalus / genetics*
  • Hydrocephalus / physiopathology
  • Hydrocephalus / therapy
  • Interleukin-10 / cerebrospinal fluid
  • Interleukin-10 / genetics
  • Interleukin-1alpha / cerebrospinal fluid
  • Interleukin-1alpha / genetics
  • Interleukin-1beta / cerebrospinal fluid
  • Interleukin-1beta / genetics
  • Interleukin-6 / cerebrospinal fluid
  • Interleukin-6 / genetics
  • Interleukin-8 / cerebrospinal fluid
  • Interleukin-8 / genetics
  • Male
  • Middle Aged
  • Prospective Studies
  • Severity of Illness Index
  • Tumor Necrosis Factor-alpha / cerebrospinal fluid
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • CCL2 protein, human
  • CXCL8 protein, human
  • Chemokine CCL2
  • IL10 protein, human
  • IL1A protein, human
  • IL1B protein, human
  • IL6 protein, human
  • Interleukin-1alpha
  • Interleukin-1beta
  • Interleukin-6
  • Interleukin-8
  • Tumor Necrosis Factor-alpha
  • Interleukin-10