Analysis of electric cigarette liquid effect on mouse brain tumor growth through EGFR and ERK activation

PLoS One. 2021 Sep 8;16(9):e0256730. doi: 10.1371/journal.pone.0256730. eCollection 2021.

Abstract

Introduction: Recently, electric cigarettes with liquid (e-liquid) were introduced as an alternative to tobacco smoking. They were promoted as possible cessation aids and were considered to be potentially less harmful than traditional tobacco-based cigarettes. However, there is little information on the toxicants present in e-liquids and their possible carcinogenic effects.

Methods: Western blot analysis was performed to identify the protein levels of cancer progression related signal transducers. Patient-derived brain tumor cells (CSC2) were injected into mouse brains and tumor growth was then observed by performing magnetic resonance imaging (MRI) and hematoxylin and eosin (H&E) staining of the whole brain. Immunohistochemistry (IHC) staining and Immunofluorescence staining were performed to study the expression of pEGFR and pERK.

Results: Western blotting revealed that e-liquids increased pEGFR and pERK expression in a dose dependent manner. Animal experiments revealed that the e-liquid treated group had accelerated tumor growth and poor prognosis compared to the vehicle group. Histological staining showed activation of pEGFR and pERK in the e-liquid treated group.

Conclusion: Our study revealed that e-liquid activates pEGFR and pERK, leading to accelerated brain tumor growth and poor prognosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Astrocytes / drug effects
  • Astrocytes / metabolism
  • Brain Neoplasms / metabolism*
  • Brain Neoplasms / pathology
  • Carcinogenesis / drug effects*
  • Cell Proliferation / drug effects
  • Cells, Cultured
  • Cigarette Smoking / metabolism
  • Disease Models, Animal
  • Electronic Nicotine Delivery Systems*
  • ErbB Receptors / metabolism
  • Extracellular Signal-Regulated MAP Kinases / metabolism*
  • Female
  • Glioblastoma / metabolism*
  • Glioblastoma / pathology
  • Heterografts / drug effects
  • Humans
  • MAP Kinase Signaling System / drug effects*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Nude
  • Neoplasm Transplantation / methods
  • Nicotine / administration & dosage*
  • Phosphorylation / drug effects
  • Prognosis
  • Propylene Glycol / administration & dosage
  • Solutions
  • Solvents / administration & dosage
  • Tumor Burden / drug effects

Substances

  • Solutions
  • Solvents
  • Propylene Glycol
  • Nicotine
  • EGFR protein, human
  • ErbB Receptors
  • Extracellular Signal-Regulated MAP Kinases

Grants and funding

The study was funded by the National Research Foundation of Korea (NRF) via the Ministry of Science and ICT in the form of research grants (NRF-2021R1A2C3013315).