Transendothelial migration induces differential migration dynamics of leukocytes in tissue matrix

J Cell Sci. 2021 Nov 1;134(21):jcs258690. doi: 10.1242/jcs.258690. Epub 2021 Nov 4.

Abstract

Leukocyte extravasation into inflamed tissue is a complex process that is difficult to capture as a whole in vitro. We employed a blood-vessel-on-a-chip model in which human endothelial cells were cultured in a tube-like lumen in a collagen-1 matrix. The vessels are leak tight, creating a barrier for molecules and leukocytes. Addition of inflammatory cytokine TNF-α (also known as TNF) caused vasoconstriction, actin remodelling and upregulation of ICAM-1. Introducing leukocytes into the vessels allowed real-time visualization of all different steps of the leukocyte transmigration cascade, including migration into the extracellular matrix. Individual cell tracking over time distinguished striking differences in migratory behaviour between T-cells and neutrophils. Neutrophils cross the endothelial layer more efficiently than T-cells, but, upon entering the matrix, neutrophils display high speed but low persistence, whereas T-cells migrate with low speed and rather linear migration. In conclusion, 3D imaging in real time of leukocyte extravasation in a vessel-on-a-chip enables detailed qualitative and quantitative analysis of different stages of the full leukocyte extravasation process in a single assay. This article has an associated First Person interview with the first authors of the paper.

Keywords: Blood vessels; Blood-vessel-on-a-chip; Endothelial cells; Extracellular matrix; Inflammation; Leukocyte transendothelial migration; Migration dynamics; Physiological hydrogel; Tissue.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Endothelial Cells*
  • Endothelium, Vascular
  • Humans
  • Leukocytes
  • Neutrophils
  • Transendothelial and Transepithelial Migration*