Abstract
HER3 is ubiquitously expressed in EGFR-mutant non-small cell lung cancer (NSCLC) irrespective of resistant mechanisms to EGFR tyrosine kinase inhibitors, thus garnering attention as a valuable therapeutic target. In this issue of Cancer Discovery, Jänne and colleagues highlight early clinical data supporting patritumab deruxtecan as a potentially appreciable agent for previously treated EGFR-mutant NSCLC.See related article by Jänne et al., p. 74.
©2022 American Association for Cancer Research.
Publication types
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Research Support, Non-U.S. Gov't
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Comment
MeSH terms
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Antibodies, Monoclonal, Humanized
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Camptothecin / analogs & derivatives
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Carcinoma, Non-Small-Cell Lung* / drug therapy
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Carcinoma, Non-Small-Cell Lung* / genetics
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ErbB Receptors / genetics
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Humans
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Lung Neoplasms* / drug therapy
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Lung Neoplasms* / genetics
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Protein Kinase Inhibitors / therapeutic use
Substances
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Antibodies, Monoclonal, Humanized
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Protein Kinase Inhibitors
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patritumab deruxtecan
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EGFR protein, human
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ErbB Receptors
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Camptothecin