Stressful events induce long-term gut microbiota dysbiosis and associated post-traumatic stress symptoms in healthcare workers fighting against COVID-19

J Affect Disord. 2022 Apr 15:303:187-195. doi: 10.1016/j.jad.2022.02.024. Epub 2022 Feb 12.

Abstract

Objective: The microbiota-gut-brain axis is a key pathway perturbed by prolonged stressors to produce brain and behavioral disorders. Frontline healthcare workers (FHWs) fighting against COVID-19 typically experience stressful event sequences and manifest some mental symptoms; however, the role of gut microbiota in such stress-induced mental problems remains unclear. We investigated the association between the psychological stress of FHW and gut microbiota.

Methods: We used full-length 16S rRNA gene sequencing to characterize the longitudinal changes in gut microbiota and investigated the impact of microbial changes on FHWs' mental status.

Results: Stressful events induced significant depression, anxiety, and stress in FHWs and disrupted the gut microbiome; gut dysbiosis persisted for at least half a year. Different microbes followed discrete trajectories during the half-year of follow-up. Microbes associated with mental health were mainly Faecalibacterium spp. and [Eubacterium] eligens group spp. with anti-inflammatory effects. Of note, the prediction model indicated that low abundance of [Eubacterium] hallii group uncultured bacterium and high abundance of Bacteroides eggerthii at Day 0 (immediately after the two-month frontline work) were significant determinants of the reappearance of post-traumatic stress symptoms in FHWs.

Limitations: The lack of metabolomic evidence and animal experiments result in the unclear mechanism of gut dysbiosis-related stress symptoms.

Conclusion: The stressful event sequences of fighting against COVID-19 induce characteristic longitudinal changes in gut microbiota, which underlies dynamic mental state changes.

Keywords: Bacteria; Frontline medical worker; Full-length 16S rRNA sequencing; Gut microbiota; Microbial dysbiosis; Stress.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • COVID-19*
  • Dysbiosis / epidemiology
  • Dysbiosis / microbiology
  • Feces / microbiology
  • Gastrointestinal Microbiome*
  • Health Personnel
  • Humans
  • RNA, Ribosomal, 16S / genetics
  • SARS-CoV-2
  • Stress Disorders, Post-Traumatic*

Substances

  • RNA, Ribosomal, 16S