Artemisinin resistance in the malaria parasite, Plasmodium falciparum, originates from its initial transcriptional response

Commun Biol. 2022 Mar 28;5(1):274. doi: 10.1038/s42003-022-03215-0.

Abstract

The emergence and spread of artemisinin-resistant Plasmodium falciparum, first in the Greater Mekong Subregion (GMS), and now in East Africa, is a major threat to global malaria elimination ambitions. To investigate the artemisinin resistance mechanism, transcriptome analysis was conducted of 577 P. falciparum isolates collected in the GMS between 2016-2018. A specific artemisinin resistance-associated transcriptional profile was identified that involves a broad but discrete set of biological functions related to proteotoxic stress, host cytoplasm remodelling, and REDOX metabolism. The artemisinin resistance-associated transcriptional profile evolved from initial transcriptional responses of susceptible parasites to artemisinin. The genetic basis for this adapted response is likely to be complex.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antimalarials* / pharmacology
  • Antimalarials* / therapeutic use
  • Artemisinins
  • Drug Resistance / genetics
  • Malaria, Falciparum* / drug therapy
  • Malaria, Falciparum* / parasitology
  • Parasites*
  • Plasmodium falciparum

Substances

  • Antimalarials
  • Artemisinins
  • artemisinin