Azithromycin promotes relapse by disrupting immune and metabolic networks after allogeneic stem cell transplantation

Blood. 2022 Dec 8;140(23):2500-2513. doi: 10.1182/blood.2022016926.

Abstract

Administration of azithromycin after allogeneic hematopoietic stem cell transplantation for hematologic malignancies has been associated with relapse in a randomized phase 3 controlled clinical trial. Studying 240 samples from patients randomized in this trial is a unique opportunity to better understand the mechanisms underlying relapse, the first cause of mortality after transplantation. We used multi-omics on patients' samples to decipher immune alterations associated with azithromycin intake and post-transplantation relapsed malignancies. Azithromycin was associated with a network of altered energy metabolism pathways and immune subsets, including T cells biased toward immunomodulatory and exhausted profiles. In vitro, azithromycin exposure inhibited T-cell cytotoxicity against tumor cells and impaired T-cell metabolism through glycolysis inhibition, down-regulation of mitochondrial genes, and up-regulation of immunomodulatory genes, notably SOCS1. These results highlight that azithromycin directly affects immune cells that favor relapse, which raises caution about long-term use of azithromycin treatment in patients at high risk of malignancies. The ALLOZITHRO trial was registered at www.clinicaltrials.gov as #NCT01959100.

Publication types

  • Randomized Controlled Trial
  • Clinical Trial, Phase III
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Azithromycin / pharmacology
  • Azithromycin / therapeutic use
  • Hematopoietic Stem Cell Transplantation*
  • Humans
  • Metabolic Networks and Pathways
  • Neoplasms*
  • Stem Cell Transplantation

Substances

  • Azithromycin

Associated data

  • ClinicalTrials.gov/NCT01959100