Early-onset venous thromboembolisms in newly diagnosed non-promyelocytic acute myeloid leukemia patients undergoing intensive induction chemotherapy

Eur J Haematol. 2023 Apr;110(4):426-434. doi: 10.1111/ejh.13920. Epub 2023 Jan 12.

Abstract

Objectives and methods: Venous thromboembolic (VTE) events are emerging as frequent complications in acute myeloid leukemia (AML); however, there is insufficient data regarding epidemiology, risk factors, and impact on outcomes. The optimal approach to balance risks of thrombosis and hemorrhage remains unclear. This retrospective single-center study in AML patients undergoing induction chemotherapy between 2007 and 2018 assessed incidence, risk factors, features, and outcomes of early-onset VTE.

Results: 423 patients (median age 59 years) were enrolled. VTE was diagnosed in 31 patients (7.3%) within 3 months of admission. The median time to VTE was 3 days. Non-central venous catheter (CVC)-related VTE occurred in 19 patients (61%). Main risk factor for VTE was leukocytosis at admission, independent of platelet counts/INR. Four patients (13%) exhibited VTE recurrence. No deaths directly related to VTE or major bleeding events associated with platelet-adjusted anticoagulation in patients with VTE were recorded. There was no clear impact of VTE on 1-year overall survival; however, non-CVC-related VTE may be associated with adverse outcomes.

Conclusions: Early-onset VTE is a common complication in newly diagnosed AML patients admitted for induction chemotherapy. Leukocytosis is an independent VTE risk factor. The potentially adverse impact of non-CVC-related VTE merits further study.

Keywords: acute myeloid leukemia; anticoagulation; induction therapy; thrombosis; venous thromboembolic event.

MeSH terms

  • Anticoagulants / adverse effects
  • Hemorrhage / drug therapy
  • Humans
  • Induction Chemotherapy
  • Leukemia, Myeloid, Acute* / drug therapy
  • Leukocytosis
  • Middle Aged
  • Retrospective Studies
  • Risk Factors
  • Venous Thromboembolism* / epidemiology
  • Venous Thrombosis* / etiology

Substances

  • Anticoagulants