Treatment efficacy in a metastatic small intestinal neuroendocrine tumour grade 2 cohort

Endocr Relat Cancer. 2023 Feb 14;30(3):e220316. doi: 10.1530/ERC-22-0316. Print 2023 Mar 1.

Abstract

Small intestinal neuroendocrine tumours (Si-NET) are often studied as a uniform group. Proliferation index Ki-67 influences prognosis and determines tumour grade. We hypothesized that Si-NET grade 2 (G2) tumours, which have a higher Ki-67 than G1 tumours, might benefit less from established treatments for metastatic disease. We conducted a retrospective cohort study of 212 patients with metastatic Si-NET G2 treated in two Swedish hospitals during 20 years (2000-2019). Median cancer-specific survival on first-line somatostatin analogues (SSA) was 77 months. Median progression-free survival (PFS) was 12.4 months when SSA was given as monotherapy and 19 months for all patients receiving first-line SSA. PFS after SSA dose escalation was 6 months in patients with radiological progression. Treatment efficacies of SSA and peptide receptor radionuclide treatment (PRRT) were studied separately in patients with Ki-67 of 3-5%, 5-10% and 10-20%. For SSA, PFS was significantly shorter at higher Ki-67 levels (31, 18 and 10 months, respectively), while there was only a minor difference in PFS for PRRT (29, 25 and 25 months). Median PFS for sequential treatment with interferon-alpha (IFNα), everolimus and chemotherapy was 6, 5 and 9 months. IFNα seemed to be effective in tumours with low somatostatin-receptor expression. In conclusion, established treatments appeared effective in Si-NET G2, despite their higher proliferation index compared to G1 tumours. However, efficacy of SSA but not PRRT was reduced at higher Ki-67 levels. SSA dose escalation provided limited disease stabilization.

Keywords: Ki-67; PRRT; Si-NET; grade 2; interferon; peptide receptor radionuclide treatment; small intestinal neuroendocrine tumours; somatostatin analogues; somatostatin receptor negative.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Humans
  • Ki-67 Antigen / metabolism
  • Neuroendocrine Tumors* / drug therapy
  • Neuroendocrine Tumors* / metabolism
  • Neuroendocrine Tumors* / radiotherapy
  • Octreotide*
  • Radioisotopes / therapeutic use
  • Retrospective Studies
  • Somatostatin / therapeutic use
  • Treatment Outcome

Substances

  • Octreotide
  • Ki-67 Antigen
  • Somatostatin
  • Radioisotopes

Supplementary concepts

  • Gastro-enteropancreatic neuroendocrine tumor