Interleukin-1 Inhibition for the Prevention and Treatment of Heart Failure

J Cardiovasc Pharmacol. 2024 Jun 1;83(6):522-530. doi: 10.1097/FJC.0000000000001497.

Abstract

Heart failure (HF) is a complex syndrome that remains a leading cause of morbidity and mortality worldwide. Abundant evidence suggests inflammation plays a key role in the development and perpetuation of HF, but there are currently no anti-inflammatory treatments approved for use in HF. Interleukin-1, the prototypical proinflammatory cytokine, has been implicated in adverse cardiac remodeling and left ventricular dysfunction. Multiple early phase clinical trials using interleukin-1 blockade in patients at risk for or diagnosed with HF have suggested favorable safety and efficacy in reducing inflammatory biomarkers, as well as positive signals in surrogate and clinical end points. Additional large scale clinical trials are urgently needed to confirm the safety and efficacy of this therapeutic approach specifically in HF. In this narrative review, we discuss current evidence regarding interleukin-1 blockade in the prevention and treatment of HF.

Publication types

  • Review
  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Anti-Inflammatory Agents / adverse effects
  • Anti-Inflammatory Agents / therapeutic use
  • Heart Failure* / drug therapy
  • Heart Failure* / immunology
  • Heart Failure* / physiopathology
  • Heart Failure* / prevention & control
  • Humans
  • Inflammation Mediators / antagonists & inhibitors
  • Inflammation Mediators / metabolism
  • Interleukin-1* / antagonists & inhibitors
  • Signal Transduction / drug effects
  • Treatment Outcome
  • Ventricular Function, Left / drug effects

Substances

  • Interleukin-1
  • Anti-Inflammatory Agents
  • Inflammation Mediators