A Population-Oriented Genetic Scoring System to Predict Phenotype: A Pathway to Personalized Medicine in Iraqis With β-Thalassemia

Hemoglobin. 2024 Mar;48(2):94-100. doi: 10.1080/03630269.2024.2319733. Epub 2024 Feb 23.

Abstract

To assess the roles of genetic modifiers in Iraqi β-thalassemia patients, and determine whether a genotype-based scoring system could be used to predict phenotype, a total of 224 Iraqi patients with molecularly characterized homozygous or compound heterozygous β-thalassemia were further investigated for α-thalassemia deletions as well as five polymorphisms namely: rs7482144 C > T at HBG2, rs1427407 G > T and rs10189857 A > G at BCL11A, and rs28384513 A > C and rs9399137 T > C at HMIP. The enrolled patients had a median age of 14 years, with 96 males and 128 females. They included 144 thalassemia major, and 80 thalassemia intermedia patients. Multivariate logistic regression analysis revealed that a model including sex and four of these genetic modifiers, namely: β+ alleles, HBG2 rs7482144, α-thalassemia deletions, and BCL11A rs1427407 could significantly predict phenotype (major versus intermedia) with an overall accuracy of 83.9%. Furthermore, a log odds genetic score based on these significant predictors had a highly significant area under curve of 0.917 (95% CI 0.882-0.953). This study underscores the notion that genetic scoring systems should be tailored to populations in question, since genetic modifiers (and/or their relative weight) vary between populations. The population-oriented genetic scoring system created by the current study to predict β-thalassemia phenotype among Iraqis may pave the way to personalized medicine in this patient's group.

Keywords: BCL11A; Genetic modifiers; HMIP; XmnI polymorphism; iraq; rs1427407; rs7482144; β-thalassemia.

MeSH terms

  • Adolescent
  • Adult
  • Alleles
  • Child
  • Child, Preschool
  • Female
  • Genotype
  • Humans
  • Iraq
  • Male
  • Phenotype*
  • Polymorphism, Single Nucleotide*
  • Precision Medicine*
  • Repressor Proteins*
  • Young Adult
  • alpha-Thalassemia / diagnosis
  • alpha-Thalassemia / genetics
  • beta-Thalassemia* / diagnosis
  • beta-Thalassemia* / genetics

Substances

  • BCL11A protein, human
  • Repressor Proteins