Immune rebalancing at the maternal-fetal interface of maternal SARS-CoV-2 infection during early pregnancy

Protein Cell. 2024 May 28;15(6):460-473. doi: 10.1093/procel/pwae006.

Abstract

The current coronavirus disease 2019 (COVID-19) pandemic caused by severe acute respiratory syndrome coronavirus (SARS-CoV-2) remains a threat to pregnant women. However, the impact of early pregnancy SARS-CoV-2 infection on the maternal-fetal interface remains poorly understood. Here, we present a comprehensive analysis of single-cell transcriptomics and metabolomics in placental samples infected with SARS-CoV-2 during early pregnancy. Compared to control placentas, SARS-CoV-2 infection elicited immune responses at the maternal-fetal interface and induced metabolic alterations in amino acid and phospholipid profiles during the initial weeks post-infection. However, subsequent immune cell activation and heightened immune tolerance in trophoblast cells established a novel dynamic equilibrium that mitigated the impact on the maternal-fetal interface. Notably, the immune response and metabolic alterations at the maternal-fetal interface exhibited a gradual decline during the second trimester. Our study underscores the adaptive immune tolerance mechanisms and establishment of immunological balance during the first two trimesters following maternal SARS-CoV-2 infection.

Keywords: SARS; fetal; immune; interface; maternal; rebalancing.

MeSH terms

  • Adult
  • COVID-19* / immunology
  • COVID-19* / virology
  • Female
  • Humans
  • Immune Tolerance
  • Placenta* / immunology
  • Placenta* / metabolism
  • Placenta* / virology
  • Pregnancy
  • Pregnancy Complications, Infectious* / immunology
  • Pregnancy Complications, Infectious* / virology
  • Pregnancy Trimester, First / immunology
  • SARS-CoV-2* / immunology
  • Transcriptome
  • Trophoblasts / immunology
  • Trophoblasts / metabolism
  • Trophoblasts / virology