Novel oral selective estrogen receptor degraders (SERDs) to target hormone receptor positive breast cancer: elacestrant as the poster-child

Expert Rev Anticancer Ther. 2024 Jun;24(6):397-405. doi: 10.1080/14737140.2024.2346188. Epub 2024 Apr 26.

Abstract

Introduction: Estrogen receptor positive (ER+) breast cancer is the most common breast cancer subtype, and therapeutic management relies primarily on inhibiting ER signaling. In the metastatic setting, ER signaling is typically targeted by selective estrogen receptor degraders (SERDs) or aromatase inhibitors (AIs), the latter of which prevent estrogen production. Activating ESR1 mutations are among the most common emergent breast cancer mutations and confer resistance to AIs.

Areas covered: Until 2023, fulvestrant was the only approved SERD; fulvestrant is administered intramuscularly, and in some cases may also have limited efficacy in the setting of certain ESR1 mutations. In 2023, the first oral SERD, elacestrant, was approved for use in ESR1-mutated, ER+/HER2- advanced breast cancer and represents a new class of therapeutic options. While the initial approval was as monotherapy, ongoing studies are evaluating elacestrant (as well as other oral SERDs) in combination with other therapies including CDK4/6 inhibitors and PI3K inhibitors, which parallels the current combination uses of fulvestrant.

Expert opinion: Elacestrant's recent approval sheds light on the use of biomarkers such as ESR1 to gauge a tumor's endocrine sensitivity. Ongoing therapeutic and correlative biomarker studies will offer new insight and expanding treatment options for patients with advanced breast cancer.

Keywords: Elacestrant; HR+ breast cancer; SERDs; endocrine therapies; targeted therapies.

Publication types

  • Review

MeSH terms

  • Administration, Oral
  • Animals
  • Aromatase Inhibitors / administration & dosage
  • Aromatase Inhibitors / pharmacology
  • Breast Neoplasms* / drug therapy
  • Breast Neoplasms* / pathology
  • Drug Resistance, Neoplasm
  • Estrogen Receptor alpha / genetics
  • Estrogen Receptor alpha / metabolism
  • Female
  • Fulvestrant / administration & dosage
  • Fulvestrant / pharmacology
  • Humans
  • Molecular Targeted Therapy
  • Mutation
  • Receptors, Estrogen / metabolism
  • Selective Estrogen Receptor Modulators / administration & dosage
  • Selective Estrogen Receptor Modulators / pharmacology
  • Signal Transduction / drug effects

Substances

  • Aromatase Inhibitors
  • Estrogen Receptor alpha
  • Fulvestrant
  • ESR1 protein, human
  • Receptors, Estrogen
  • Selective Estrogen Receptor Modulators