SMAD4 mosaicism in juvenile polyposis: Essential contribution of somatic analysis in diagnosis

Am J Med Genet A. 2024 Sep;194(9):e63648. doi: 10.1002/ajmg.a.63648. Epub 2024 May 2.

Abstract

Juvenile polyposis syndrome (JPS) is a rare disease characterized by multiple hamartomatous polyps in the gastrointestinal tract, associated with pathogenic variants of BMPR1A and SMAD4. We present the description of SMAD4 mosaicism in a 30-year-old man who had caecum adenocarcinoma, 11 juvenile colon polyps and epistaxis since childhood. We conducted NGS polyposis and CRC panel analysis on DNA extracted from two polyps, revealing a likely pathogenic SMAD4 variant: NM_005359.5:c. 1600C>T, p.(Gln534*). This variant was then identified at a very low frequency on blood and normal colonic tissue, by targeted visualization of previously obtained NGS data. These findings support the presence of a likely pathogenic mosaic SMAD4 variant that aligns with the patient's phenotype. Given the relatively frequent occurrence of de novo SMAD4 mutations, somatic mosaicism could account for a significant proportion of sporadic JPS patients with unidentified pathogenic variants. This case underscores the diagnosis challenge of detecting mosaicism and emphasizes the importance of somatic analyses.

Keywords: Mosaicism; SMAD4; juvenile polyposis syndrome.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Bone Morphogenetic Protein Receptors, Type I / genetics
  • High-Throughput Nucleotide Sequencing
  • Humans
  • Intestinal Polyposis* / congenital
  • Intestinal Polyposis* / diagnosis
  • Intestinal Polyposis* / genetics
  • Male
  • Mosaicism*
  • Mutation
  • Neoplastic Syndromes, Hereditary* / diagnosis
  • Neoplastic Syndromes, Hereditary* / genetics
  • Phenotype
  • Smad4 Protein* / genetics

Substances

  • BMPR1A protein, human
  • Bone Morphogenetic Protein Receptors, Type I
  • Smad4 Protein
  • SMAD4 protein, human

Supplementary concepts

  • Juvenile polyposis syndrome