Synaptic Protein Loss in Extracellular Vesicles Reflects Brain and Retinal Atrophy in People With Multiple Sclerosis

Neurol Neuroimmunol Neuroinflamm. 2024 Jul;11(4):e200257. doi: 10.1212/NXI.0000000000200257. Epub 2024 May 16.

Abstract

Objectives: To assess whether the rate of change in synaptic proteins isolated from neuronally enriched extracellular vesicles (NEVs) is associated with brain and retinal atrophy in people with multiple sclerosis (MS).

Methods: People with MS were followed with serial blood draws, MRI (MRI), and optical coherence tomography (OCT) scans. NEVs were immunocaptured from plasma, and synaptopodin and synaptophysin proteins were measured using ELISA. Subject-specific rates of change in synaptic proteins, as well as brain and retinal atrophy, were determined and correlated.

Results: A total of 50 people with MS were included, 46 of whom had MRI and 45 had OCT serially. The rate of change in NEV synaptopodin was associated with whole brain (rho = 0.31; p = 0.04), cortical gray matter (rho = 0.34; p = 0.03), peripapillary retinal nerve fiber layer (rho = 0.37; p = 0.01), and ganglion cell/inner plexiform layer (rho = 0.41; p = 0.006) atrophy. The rate of change in NEV synaptophysin was also correlated with whole brain (rho = 0.31; p = 0.04) and cortical gray matter (rho = 0.31; p = 0.049) atrophy.

Discussion: NEV-derived synaptic proteins likely reflect neurodegeneration and may provide additional circulating biomarkers for disease progression in MS.

MeSH terms

  • Adult
  • Atrophy*
  • Brain* / diagnostic imaging
  • Brain* / metabolism
  • Brain* / pathology
  • Extracellular Vesicles* / metabolism
  • Female
  • Humans
  • Magnetic Resonance Imaging
  • Male
  • Microfilament Proteins / metabolism
  • Middle Aged
  • Multiple Sclerosis* / diagnostic imaging
  • Multiple Sclerosis* / metabolism
  • Multiple Sclerosis* / pathology
  • Retina* / diagnostic imaging
  • Retina* / metabolism
  • Retina* / pathology
  • Synaptophysin* / metabolism
  • Tomography, Optical Coherence