Fatal ataxic encephalopathy and carnitine acetyltransferase deficiency: a functional defect of pyruvate oxidation?

Neurology. 1979 Dec;29(12):1578-83. doi: 10.1212/wnl.29.12.1578.

Abstract

A 3-year 8-month-old girl died after 14 months of illness characterized by episodes of intermittent ataxia associated with oculomotor palsy, hypotonia, mental confusion, and disturbances of consciousness. In the last 4 months of life, there were signs of liver dysfunction. Pyruvate dehydrogenase and alpha-ketoglutarate dehydrogenase activities were normal in autopsy brain specimens and in cultured fibroblasts from the patient. Carnitine acetyltransferase was deficient in liver, brain, kidney, and cultured fibroblasts. Medium- and long-chain carnitine acyltransferase activities were normal. It is proposed that a functional defect of acetyl-coenzyme A (acetyl-CoA) utilization in brain mitochondria accompanies the carnitine acetyltransferase deficiency.

Publication types

  • Case Reports

MeSH terms

  • Acetyltransferases / deficiency*
  • Alanine Transaminase / blood
  • Aspartate Aminotransferases / blood
  • Ataxia / enzymology*
  • Brain / enzymology
  • Carnitine O-Acetyltransferase / deficiency*
  • Child, Preschool
  • Dihydrolipoamide Dehydrogenase / metabolism
  • Female
  • Fibroblasts / enzymology
  • Humans
  • Ketoglutarate Dehydrogenase Complex / metabolism
  • Liver / enzymology
  • Oxidation-Reduction
  • Pyruvate Dehydrogenase Complex / metabolism
  • Pyruvates / metabolism*

Substances

  • Pyruvate Dehydrogenase Complex
  • Pyruvates
  • Ketoglutarate Dehydrogenase Complex
  • Dihydrolipoamide Dehydrogenase
  • Acetyltransferases
  • Carnitine O-Acetyltransferase
  • Aspartate Aminotransferases
  • Alanine Transaminase