S-adenosyl-L-methionine antagonizes ethynylestradiol-induced bile cholesterol supersaturation in humans without modifying the estrogen plasma kinetics

Gastroenterology. 1982 Feb;82(2):223-7.

Abstract

Previous investigations have shown that S-adenosyl-L-methionine antagonizes both cholestasis and bile lipid alterations induced by ethynylestradiol in the rat. Since it is still unknown whether S-adenosyl-L-methionine prevents ethynylestradiol-induced cholesterol supersaturation of bile in humans also and how methynation interferes with plasma ethynylestradiol disappearance, the present study has been carried out. On different days, 5 nonobese subjects with indwelling biliary drainage and reestablished enterohepatic bile circulation received 200 micrograms of intravenous ethynylestradiol or ethynylestradiol plus 200 mg of S-adenosyl-L-methionine intramuscularly. S-adenosyl-L-methionine administration prevented any ethynylestradiol-induced changes in hepatic bile saturation index. Moreover, S-adenosyl-L-methionine did not affect ethylnylestradiol pharmacokinetics which was evaluated in a group of 6 nonobese women receiving 100 micrograms of intravenous ethynylestradiol or ethynylestradiol plus 100 mg of S-adenosyl-L-methionine intramuscularly. These findings indicate that S-adenosyl-L-methionine prevents ethynylestradiol-induced bile lipid changes without interfering with ethynylestradiol kinetics in humans.

MeSH terms

  • Adult
  • Bile / drug effects*
  • Cholesterol / metabolism*
  • Ethinyl Estradiol / antagonists & inhibitors
  • Ethinyl Estradiol / metabolism*
  • Female
  • Humans
  • Kinetics
  • Lipid Metabolism
  • Male
  • Methylation
  • Middle Aged
  • S-Adenosylmethionine / pharmacology*

Substances

  • Ethinyl Estradiol
  • S-Adenosylmethionine
  • Cholesterol