[Dysfunction of E-cadherin due to mutation of beta-catenin in a scirrhous gastric cancer cell line]

Nihon Rinsho. 1995 Jul;53(7):1590-4.
[Article in Japanese]

Abstract

E-cadherin is a calcium dependent cell-cell adhesion molecule. In cancer tissue, detachment of the adhesion system is indispensable for invasion and metastasis of cancer cells. We have investigated mechanism of the dysfunction of E-cadherin-dependent cell-cell adhesion system in gastric carcinoma cells. Although the high expression of E-cadherin in a scirrhous gastric cancer cell line HSC-39, the function of E-cadherin was completely abolished. Western blotting of beta-catenin in HSC-39 cells demonstrated that a truncated beta-catenin was detected. The truncation was due to partial deletion of beta-catenin DNA. It was concluded that in HSC-39 loss of E-cadherin dependent cell-cell adhesion was due to mutation of beta-catenin gene.

Publication types

  • English Abstract
  • Review

MeSH terms

  • Adenocarcinoma, Scirrhous / genetics
  • Adenocarcinoma, Scirrhous / pathology*
  • Cadherins / genetics*
  • Cytoskeletal Proteins / genetics*
  • Gene Deletion
  • Humans
  • Mutation*
  • Stomach Neoplasms / genetics
  • Stomach Neoplasms / pathology*
  • Trans-Activators*
  • Tumor Cells, Cultured
  • beta Catenin

Substances

  • CTNNB1 protein, human
  • Cadherins
  • Cytoskeletal Proteins
  • Trans-Activators
  • beta Catenin