Role of local pancreatic blood flow in development of hemorrhagic pancreatitis induced by stress in rats

Pancreas. 1993 Jul;8(4):499-505. doi: 10.1097/00006676-199307000-00015.

Abstract

Our previous data showed that the pancreatitis induced in rats by cerulein develops into hemorrhagic pancreatitis following water-immersion stress. The present study examined the effects of water-immersion stress and high doses of cerulein (intraperitoneal injection) on pancreatic blood flow. Five hours of water-immersion stress reduced the local pancreatic blood flow to approximately 30% of the initial value (253.75 +/- 12.58 ml/min/100 g) without causing any histological alterations. Blood flow was decreased as early as 1 h after the immersion and reached the lowest value (30% of initial value) 3 h after the immersion. The administration of 40 micrograms/kg body wt cerulein as two intraperitoneal injections reduced the pancreatic blood flow by 40% 5 h after the first cerulein injection. The injections of cerulein combined with water-immersion stress did not reduce the pancreatic blood flow more than did water-immersion stress alone. The systemic blood pressure was unaffected during 5 h of water immersion after the cerulein injections. These findings suggest that in rats the stress-induced decrease of local pancreatic blood flow may not produce pancreatitis, but may aggravate an existing acute pancreatitis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amylases / blood
  • Animals
  • Capillaries / pathology
  • Dopamine / pharmacology
  • Endothelium, Vascular / pathology
  • Hemorrhage / etiology
  • Hemorrhage / pathology
  • Hemorrhage / physiopathology*
  • Male
  • Organ Size / physiology
  • Pancreas / blood supply*
  • Pancreas / pathology
  • Pancreatitis / etiology
  • Pancreatitis / pathology
  • Pancreatitis / physiopathology*
  • Rats
  • Rats, Sprague-Dawley
  • Regional Blood Flow / drug effects
  • Regional Blood Flow / physiology
  • Stress, Physiological / physiopathology*
  • Thrombosis / etiology
  • Thrombosis / pathology
  • Vasopressins / pharmacology

Substances

  • Vasopressins
  • Amylases
  • Dopamine