Cytotoxic and proliferative T cell responses in HIV-1-infected Macaca nemestrina

J Clin Invest. 1995 Jan;95(1):248-56. doi: 10.1172/JCI117647.

Abstract

Macaca nemestrina has been described as an animal model for acute HIV-1 infection. This animal, unlike most infected humans, appears to contain HIV-1 replication. Therefore analysis of HIV-1-specific proliferative and cytotoxic T lymphocyte (CTL) responses following HIV-1 challenge of M. nemestrina may provide information into the role of such responses in both the control of acute HIV infection and protective immunity. Although CD4+ T cell responses to HIV-1 are generally difficult to detect in HIV-1-infected humans, early and persistent CD4+ T cell proliferative responses to HIV-1 antigens were detected in all HIV-1-inoculated M. nemestrina. HIV-1-specific CD8+ CTL responses were evaluated in PBMC by stimulation with autologous cells expressing HIV-1 genes, limiting dilution precursor frequency analysis, and T cell cloning. CTL reactive with gag, env, and nef were present 4-8 wk after infection, and persisted to 140 wk after infection. The presence of both CD4+ and CD8+ T cell responses before and after clearance of HIV-1 viremia is consistent with a role for these responses in the successful control of HIV-1 viral replication observed in M. nemestrina. Further studies of T cell immunity in these animals that resist disease should provide insights into the immunobiology of HIV-1 infection.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / immunology
  • Cells, Cultured
  • Clone Cells
  • Cytotoxicity, Immunologic
  • Disease Models, Animal
  • Gene Products, env / immunology
  • Gene Products, gag / immunology
  • Gene Products, nef / immunology
  • HIV Infections / immunology*
  • HIV-1 / immunology*
  • Lymphocyte Activation
  • Macaca nemestrina / immunology*
  • Macaca nemestrina / virology
  • T-Lymphocytes / immunology*
  • T-Lymphocytes, Regulatory / immunology
  • Time Factors
  • nef Gene Products, Human Immunodeficiency Virus

Substances

  • Gene Products, env
  • Gene Products, gag
  • Gene Products, nef
  • nef Gene Products, Human Immunodeficiency Virus