Neurofibromatosis type 2 (NF2) gene is somatically mutated in mesothelioma but not in lung cancer

Cancer Res. 1995 Mar 15;55(6):1227-31.

Abstract

We have found 16 of 28 small cell lung cancers, 17 of 31 non-small cell lung cancers, 2 of 3 carcinoids, and 12 of 14 mesotheliomas that had chromosome 22 cytogenetic abnormalities. To determine whether the neurofibromatosis type 2 (NF2) gene located on chromosome 22 participates in the oncogenesis of these malignancies, we studied DNAs from lung cancer cell lines and mesotheliomas using Southern blot analysis and the single-strand conformation polymorphism (SSCP) technique for mutations covering 8 of the 16 known NF2 exons. We detected 7 mutations in 17 mesotheliomas (41%) within the coding region of NF2 but none in 75 lung cancer cell lines (38 small cell lung cancers, 34 non-small cell lung cancers, and 3 carcinoids). These mutations were found to be somatic when normal tissue was available for testing. Four mesothelioma cell lines had relatively large deletions (approximately 10-50 kilobases) in the NF2 gene detectable by Southern blot analysis. Two mesothelioma cell lines had nonsense mutations at codons 57 and 341, respectively. Another mesothelioma obtained as a specimen directly from a patient, had a 10-base pair microdeletion from nucleotide 1004 to nucleotide 1013 causing a frameshift mutation. These results suggest that the NF2 gene participates in the oncogenesis in a subset of mesotheliomas but not in lung cancers.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Base Sequence
  • Chromosomes, Human, Pair 22
  • DNA-Binding Proteins / genetics
  • Gene Deletion
  • Genes, Neurofibromatosis 2*
  • Genes, Tumor Suppressor
  • Humans
  • Lung Neoplasms / genetics*
  • Mesothelioma / genetics*
  • Molecular Sequence Data
  • Mutation*
  • Transcription Factors / genetics
  • Tumor Cells, Cultured
  • WT1 Proteins

Substances

  • DNA-Binding Proteins
  • Transcription Factors
  • WT1 Proteins