The effects of chemotherapy on morphology, cellular proliferation, apoptosis and oncoprotein expression in primary breast carcinoma

Br J Cancer. 1994 Aug;70(2):335-41. doi: 10.1038/bjc.1994.303.

Abstract

The use of chemotherapy as a form of primary treatment for breast cancer is increasing and, as a result, more resection specimens contain tumours which have been exposed to cytotoxic drugs. We have studied the effects of chemotherapy on the tumour morphology and various biological features of breast carcinoma in a group of 35 patients. These were a group who responded to treatment in a clinical study of the use of primary chemotherapy designed to reduce tumour bulk prior to surgery. Characteristic morphological changes, temporally related to the administration of cytotoxic agents, are seen. The malignant cells become enlarged with vacuolated cytoplasm and vesicular nuclei containing prominent nuclei; occasionally the nuclei were angular and hyperchromatic. These features are interpreted as degenerative in nature. In 15 cases sufficient material was present in the pretreatment biopsies to compare the grade of the tumours before and after chemotherapy: changes were found in six tumours. Cytotoxic drugs do not induce a consistent pattern of change in the proliferation and apoptotic indices of individual tumours, but there is a tendency to reduce proliferative activity over all the tumours as a group. It was also found that chemotherapy is capable of modifying the expression of the oncoproteins c-erbB-2 and p53 in a minority of cases of breast cancer, usually resulting in an acquisition of immunoreactive oncoprotein. It is important to be aware of these effects when studying breast carcinomas removed after chemotherapy.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Aged
  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use*
  • Apoptosis / drug effects*
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / pathology*
  • Breast Neoplasms / surgery
  • Cell Division / drug effects
  • Cyclophosphamide / administration & dosage
  • Doxorubicin / administration & dosage
  • Epirubicin / administration & dosage
  • ErbB Receptors / analysis
  • Feasibility Studies
  • Female
  • Fluorouracil / administration & dosage
  • Humans
  • Immunohistochemistry
  • Middle Aged
  • Neoplasm Proteins / analysis*
  • Oncogene Proteins / analysis*
  • Proto-Oncogene Proteins / analysis
  • Receptor, ErbB-2
  • Tumor Suppressor Protein p53 / analysis

Substances

  • Neoplasm Proteins
  • Oncogene Proteins
  • Proto-Oncogene Proteins
  • Tumor Suppressor Protein p53
  • Epirubicin
  • Doxorubicin
  • Cyclophosphamide
  • ErbB Receptors
  • Receptor, ErbB-2
  • Fluorouracil