Male hypogonadism in myotonic dystrophy is related to (CTG)n triplet mutation

J Endocrinol Invest. 1994 May;17(5):381-3. doi: 10.1007/BF03349005.

Abstract

The Authors considered the relationship between hypogonadism in myotonic dystrophy (MD) and MT-PK gene mutation. Twenty-seven subjects were studied, and the (CTG)n amplification varied from 70 to 1520 (mean 661 +/- 463). Hypergonadotropic-hypogonadism with LH levels of 6.94 +/- 3.87 and FSH 14.54 +/- 9.58 IU/L was present; testosterone still showed normal values (505.7 +/- 376.2 ng/dl), but 44.4% of patients had abnormal serum level less than 250 ng/dl. We found a significant correlation (p < 0.001) between CTG repeat size and levels of both LH and FSH: these findings suggest that the severity of hypogonadism is related to MT-PK gene mutation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Base Sequence
  • Humans
  • Hypogonadism / etiology*
  • Hypogonadism / genetics*
  • Male
  • Middle Aged
  • Molecular Sequence Data
  • Mutation
  • Myotonic Dystrophy / complications*
  • Myotonic Dystrophy / enzymology
  • Myotonin-Protein Kinase
  • Protein Kinases / genetics
  • Protein Serine-Threonine Kinases / genetics
  • Repetitive Sequences, Nucleic Acid

Substances

  • DMPK protein, human
  • Protein Kinases
  • Myotonin-Protein Kinase
  • Protein Serine-Threonine Kinases

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