Interaction of activin A and gonadal steroids on FSH secretion from primary cultured rat anterior pituitary cells

Biochem Biophys Res Commun. 1993 Jul 15;194(1):413-9. doi: 10.1006/bbrc.1993.1835.

Abstract

To clarify the mechanism of FSH secretion from the pituitary induced by activin A, we studied the interaction between activin A and gonadal steroids in inducing FSH release from primary cultured female rat pituitary cells in serum-free medium. The basal release of FSH was stimulated by activin A, testosterone (T) and progesterone (P), and T and P also facilitated basal FSH release stimulated by activin A. The GnRH-stimulated FSH release was facilitated by activin A, P and 17 beta-estradiol (E2), but suppressed by T. The effect of activin A on GnRH-stimulated FSH release was facilitated by P, but not affected by T or E2. These findings suggested that the interaction between activin A and T or P may be involved in the regulation of FSH secretion during the estrus cycle in rats.

MeSH terms

  • Activins
  • Analysis of Variance
  • Animals
  • Cells, Cultured
  • Dose-Response Relationship, Drug
  • Drug Interactions
  • Estradiol / pharmacology*
  • Female
  • Follicle Stimulating Hormone / metabolism*
  • Gonadotropin-Releasing Hormone / pharmacology
  • Growth Substances / pharmacology
  • Inhibins / pharmacology*
  • Kinetics
  • Pituitary Gland, Anterior / drug effects
  • Pituitary Gland, Anterior / metabolism*
  • Progesterone / pharmacology*
  • Rats
  • Rats, Wistar
  • Recombinant Proteins / pharmacology*
  • Testosterone / pharmacology*

Substances

  • Growth Substances
  • Recombinant Proteins
  • Activins
  • Gonadotropin-Releasing Hormone
  • Testosterone
  • Progesterone
  • Estradiol
  • Inhibins
  • Follicle Stimulating Hormone