Repression of stromelysin metalloprotease expression in rat fibrosarcoma cells by dimethylsulfoxide

Clin Exp Metastasis. 1993 Jan;11(1):77-82. doi: 10.1007/BF00880068.

Abstract

Metalloproteases are implicated in conferring invasive properties to tumor cells. We show here that treatment of ras-oncogene-transformed rat fibroblasts with dimethylsulfoxide (DMSO) results in a reversible decrease in stromelysin mRNA. Furthermore, stromelysin expression was found to be repressed by DMSO, but not by glucocorticoid hormone, in a fibrosarcoma cell line showing low AP-1 (fos/jun) transcription factor activity. In two fibrosarcoma cell lines which express high levels of stromelysin and low levels of 68 kDa type IV collagenase, the DMSO-induced decrease in stromelysin expression was paralleled by a decreased invasive propensity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Northern
  • Cell Adhesion / drug effects
  • Cell Division / drug effects
  • Cell Line, Transformed
  • Cell Movement / drug effects
  • Dimethyl Sulfoxide / pharmacology*
  • Enzyme Repression / drug effects
  • Fibrosarcoma / enzymology*
  • Fibrosarcoma / pathology
  • Matrix Metalloproteinase 3
  • Metalloendopeptidases / biosynthesis
  • Metalloendopeptidases / drug effects*
  • Neoplasm Invasiveness / physiopathology
  • Rats
  • Tumor Cells, Cultured / drug effects

Substances

  • Metalloendopeptidases
  • Matrix Metalloproteinase 3
  • Dimethyl Sulfoxide