The three processes leading to post PTCA restenosis: dependence on the lesion substrate

Thromb Haemost. 1995 Jul;74(1):552-9.

Authors

V Fuster¹, E Falk, J T Fallon, L Badimon, J H Chesebro, J J Badimon

Affiliation

¹ Cardiovascular Biology Research Laboratory, Cardiovascular Institute Mount Sinai School of Medicine, New York, NY 10029, USA.

PMID: 8578523

No abstract available

Publication types

Review

MeSH terms

Angioplasty, Balloon, Coronary / adverse effects*
Arteriosclerosis
Coronary Artery Disease / pathology
Coronary Artery Disease / prevention & control
Coronary Artery Disease / therapy*
Drug Delivery Systems
Endothelium, Vascular / pathology
Extracellular Matrix / metabolism
Fibrinolytic Agents / therapeutic use
Gene Expression Regulation
Growth Substances / physiology
Heart Diseases / etiology
Heart Diseases / therapy
Humans
Macrophages / pathology
Models, Biological
Muscle, Smooth, Vascular / pathology
Recurrence
Stents
Thrombin / metabolism
Thrombosis / etiology
Thrombosis / therapy

Substances

Fibrinolytic Agents
Growth Substances
Thrombin