Relationship between cAMP induced inhibition of human meningioma cell proliferation and autocrine secretion of interleukin-6

Life Sci. 1996;58(16):1323-9. doi: 10.1016/0024-3205(96)00098-7.

Abstract

Previous studies have suggested that activation of the adenylyl cyclase - cAMP system in meningiomas results in decreased mitosis. We have used meningioma cell culture to further investigate this phenomenon and to examine the potential role played by interleukin-6 (IL6). Incubation of cultured meningioma cells for 4-6 days with cholera toxin and theophylline, both of which increase intracellular cAMP levels, markedly stimulated IL6 secretion and inhibited cell growth rate. Similar effects were observed with 8-bromo-cAMP. In contrast, a neutralising polyclonal antibody against IL6 significantly stimulated meningioma proliferation and reduced the inhibitory effects of 8-bromo-cAMP. These results support the concept that IL6 acts as an autocrine / paracrine inhibitory factor for meningioma proliferation, and that the inhibition exerted by elevated intracellular cAMP levels may be at least partially mediated via increased secretion of the cytokine.

MeSH terms

  • 8-Bromo Cyclic Adenosine Monophosphate / pharmacology
  • Adult
  • Aged
  • Antibodies / pharmacology
  • Cell Division / drug effects
  • Cell Division / physiology
  • Cyclic AMP / biosynthesis
  • Cyclic AMP / pharmacology*
  • Female
  • Humans
  • Interleukin-6 / metabolism*
  • Male
  • Meningioma / metabolism*
  • Meningioma / pathology*
  • Middle Aged
  • Neutralization Tests
  • Theophylline / pharmacology
  • Tumor Cells, Cultured / drug effects

Substances

  • Antibodies
  • Interleukin-6
  • 8-Bromo Cyclic Adenosine Monophosphate
  • Theophylline
  • Cyclic AMP