Extracellular ubiquitin regulates the growth of human hematopoietic cells

Biochem Biophys Res Commun. 1996 Jun 14;223(2):226-8. doi: 10.1006/bbrc.1996.0875.

Abstract

We investigated the effects of extracellular ubiquitin on the colony formation of human hematopoietic progenitor cells (CFU-GM, CFU-E and BFU-E) and a granulocyte-colony stimulating factor (G-CSF)-dependent human myeloblastic leukemic cell line, OCI/AML/1a. Ubiquitin exhibited inhibitory effects on CFU-GM, CFU-E, BFU-E and OCI/AML/1a colony formation. Extracellular ubiquitin also inhibited the growth of KT3 (T lymphoblast), K562 (erythroleukemia) and Daudi (Burkitt's lymphoma) cells, stimulated the growth of HL60 (promyelocytic leukemia) and Jurkat (T-ALL) cells and showed no effect on the growth of U937 cells (monocytic leukemia). These results indicate that another, previously unrecognized function of extracellular ubiquitin is to regulate hematopoiesis.

MeSH terms

  • Burkitt Lymphoma
  • Cell Division / drug effects
  • Cell Line
  • Colony-Forming Units Assay
  • Granulocyte Colony-Stimulating Factor / pharmacology
  • HL-60 Cells
  • Hematopoietic Stem Cells / cytology*
  • Hematopoietic Stem Cells / drug effects
  • Humans
  • Interleukin-3 / pharmacology
  • Interleukin-6 / pharmacology
  • Leukemia
  • Leukemia, Erythroblastic, Acute
  • Recombinant Proteins / pharmacology
  • Tumor Cells, Cultured
  • Ubiquitins / pharmacology*

Substances

  • Interleukin-3
  • Interleukin-6
  • Recombinant Proteins
  • Ubiquitins
  • Granulocyte Colony-Stimulating Factor