Induction of differentiation of embryonal carcinoma F9 cells by iron chelators

Res Commun Mol Pathol Pharmacol. 1995 Nov;90(2):211-20.

Abstract

The effects of several chelators, including hinokitiol, on embryonal carcinoma F9 cell differentiation were assessed by assaying the production of plasminogen activator (PA) as a differentiation marker protein. Hinokitiol and tropolone which were potent differentiation inducers lost their activity following preincubation with Fe2+ and Fe3+ ions. Other metal ions had no or little effect on the hinokitiol-induced differentiation. Of several chelators examined, dithizone induced differentiation as effectively as did hinokitiol and tropolone. Dithizone-induced differentiation was also inhibited by preincubation with Fe3+ ions. It was concluded that some potent iron chelators could trigger the teratocarcinoma F9 cells to differentiate through the chelation with intracellular iron ions.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Anticarcinogenic Agents / pharmacology
  • Carcinoma, Embryonal / pathology*
  • Cations
  • Cell Differentiation / drug effects
  • Iron Chelating Agents / pharmacology*
  • Kinetics
  • Metals / pharmacology
  • Mice
  • Monoterpenes*
  • Teratocarcinoma / pathology
  • Tropolone / analogs & derivatives
  • Tropolone / pharmacology
  • Tumor Cells, Cultured / drug effects

Substances

  • Anticarcinogenic Agents
  • Cations
  • Iron Chelating Agents
  • Metals
  • Monoterpenes
  • Tropolone
  • beta-thujaplicin