Abstract
IL13 induces the same biological effects as IL4 in normal human B cells. We show that as in the IL4R complex, both IL4R alpha and IL2R gamma c are components of the IL13R and that both cytokines induced STAT6, STAT3 and STAT5 activation in B cells. In spite of this similar downstream signalling, IL4 and IL13 used a different set of Janus kinases: IL13 is unable to activate JAK1 and JAK3.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antigens, CD / chemistry
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Antigens, CD / metabolism*
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B-Lymphocytes / immunology
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B-Lymphocytes / metabolism*
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Child
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DNA-Binding Proteins / metabolism*
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Humans
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Immunoglobulin Constant Regions / immunology
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Immunoglobulin gamma-Chains / immunology
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Interleukin-13 / metabolism
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Interleukin-13 Receptor alpha1 Subunit
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Interleukin-4 / metabolism
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Janus Kinase 1
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Janus Kinase 3
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Milk Proteins*
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Phosphorylation
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Protein-Tyrosine Kinases / metabolism
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Receptors, Interleukin / chemistry
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Receptors, Interleukin / metabolism*
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Receptors, Interleukin-13
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Receptors, Interleukin-2 / immunology
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Receptors, Interleukin-4
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Recombinant Proteins / metabolism
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STAT3 Transcription Factor
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STAT5 Transcription Factor
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STAT6 Transcription Factor
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Time Factors
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Trans-Activators / metabolism*
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Tyrosine / metabolism
Substances
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Antigens, CD
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DNA-Binding Proteins
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IL13RA1 protein, human
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Immunoglobulin Constant Regions
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Immunoglobulin gamma-Chains
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Interleukin-13
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Interleukin-13 Receptor alpha1 Subunit
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Milk Proteins
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Receptors, Interleukin
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Receptors, Interleukin-13
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Receptors, Interleukin-2
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Receptors, Interleukin-4
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Recombinant Proteins
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STAT3 Transcription Factor
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STAT3 protein, human
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STAT5 Transcription Factor
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STAT6 Transcription Factor
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STAT6 protein, human
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Trans-Activators
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Interleukin-4
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Tyrosine
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Protein-Tyrosine Kinases
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JAK1 protein, human
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JAK3 protein, human
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Janus Kinase 1
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Janus Kinase 3