Regulation of acetyl coenzyme-A carboxylase gene in a transgenic animal model

Biochem Biophys Res Commun. 1996 Oct 14;227(2):547-52. doi: 10.1006/bbrc.1996.1543.

Abstract

The gene for acetyl-CoA carboxylase, the rate-limiting enzyme in the biosynthesis of long-chain fatty acids, contains two promoters; promoter I (PI) and promoter II (PII) which are responsible for generation of class I and class II ACC mRNAs, respectively. Class I ACC mRNAs are present in adipose tissue, but only a trace was found in the liver under normal physiological conditions. However, class I mRNAs were induced under stimulated lipogenic conditions. To investigate how PI is regulated in vivo, we generated transgenic mice containing a reporter gene under the control of PI. In transgenic mice, PI is generally inactive and a small amount of PI activity was found only in the adipose tissues of female animals. Stimulated lipogenic conditions activated PI about 17-fold over normal conditions and again only in white adipose tissues of female animals.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acetyl-CoA Carboxylase / biosynthesis*
  • Acetyl-CoA Carboxylase / genetics*
  • Adipose Tissue / enzymology*
  • Animals
  • Chloramphenicol O-Acetyltransferase / biosynthesis
  • Female
  • Gene Expression Regulation, Enzymologic*
  • Genes, Reporter
  • Liver / enzymology
  • Male
  • Mice
  • Mice, Inbred Strains
  • Mice, Transgenic
  • Organ Specificity
  • Promoter Regions, Genetic*
  • Pseudopregnancy
  • RNA, Messenger / biosynthesis
  • Restriction Mapping
  • Transcription, Genetic

Substances

  • RNA, Messenger
  • Chloramphenicol O-Acetyltransferase
  • Acetyl-CoA Carboxylase